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Contribution produced by the fundamental analysis on neuronal chloride Previous work carried out by the team of experts led by Yehezkel Ben-Ari in INSERM unit 901, the Institut de Neurobiologie de la M-diterran-e in Marseille, on intracellular chloride concentrations possess demonstrated they are abnormally high in immature neurons or neurons previously affected by epileptic seizures or other cerebral lesions. Many anxiolytics, analgesics and antiepileptics work by increasing the effects of GABA – the mind's main chemical mediator – which inhibits the neurons. When the cells include a high chloride concentration, nevertheless, GABA's effects are reversed. GABA simply no inhibits the neurons much longer; the anxiolytic molecules instead accentuate these effects.